MAML1 and hyperinsulinemic hypoglycemia, familial, 4: Similar to that of the slim1 mutant, transcriptomic analysis of the q‐lsu‐KO mutant under [nS] conditions revealed altered expression of genes that are markers of S deficiency, for example, SULTR1;1, SULTR2;1, APR3, and GGCT2;1, and genes involved in GSL synthesis and degradation, particularly aliphatic GSL known for their sensitivity to S deprivation (Maruyama‐Nakashita, 2017), such as SDI1, BGLU28, MAM1, BCAT4, and CYP79F1.