These improvements were also reflected in our results, where significant decreases in ALT and AST were observed alongside improvements in steatosis and inflammation. However, similar to our review, the effect of vitamin E on fibrosis was limited, which aligns with the findings of both Usman et al. and Perumpail et al., who highlighted the minimal impact on fibrosis [7,26]. The gene discussed is GPT; the disease is steatosis.