We found that when we infect iBMDMs lacking TLR9 with cidal treated bacteria, we observe diminished TNF signaling relative to the corresponding infections of WT iBMDMs, whereas signaling from static-treated bacteria was maintained (if anything we observe a slight increase in static-treated TNF signal in the Tlr9−/− iBMDMs relative to WT iBMDMs). Here, TNF is linked to infection.