In contrast, overexpression of circHipk3 in C57BL/6J mice stimulated by Ang II promoted AAA formation in comparison with the AAV‐mScarlet group (Figure 4A), whereas ultrasound imaging (Figure 4B), AAA incidence (Figure 4C), the rupture rate of AAA (Figure 4D) and maximum aortic diameter (Figure 4E), which reflects the severity of AAA lesions was increased in the AAV‐mScarlet group. Here, AGT is linked to triple-A syndrome.