Another study showed that plasma cell-like DCs were significantly elevated in the acute phase of GBS, and their levels were positively correlated with the severity of disease in GBS patients, and the expression of TLR9 and surface co-stimulatory molecules were significantly elevated in plasma cell-like DCs, suggesting that plasma cell-like DCs are involved in the pathogenesis of GBS (32). Here, TLR9 is linked to Guillain-Barre syndrome.