Lactobacillus casei also reduced hyperglycaemia via bile acid (BA)—chloride exchange, which involves the upregulation of Chloride Channels 1 to 7 (ClC1-7), Glycine Receptor Alpha 1 (GlyRa1), Solute Carrier Family 26 Member 3 (SLC26A3), Solute Carrier Family 26 Member 6 (SLC26A6), Gamma-Aminobutyric Acid Type A Receptor Alpha 1 (GABAAa1), Bestrophin 3 (BEST3), and Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) [78]. Here, SLC26A6 is linked to Hyperglycemia.