Although AMQ exhibits modest potency as an antidote for OPC poisoning, reported results have demonstrated AMQ’s capacity for interaction with amino acid residues of the AChE active site and, consequently, the potency of AMQ to act as a reversible inhibitor of AChE [20,21], a feature highly appreciated in the development of drugs for the symptomatic treatment of AD. The gene discussed is ACHE; the disease is Alzheimer disease.