Jin et al. demonstrated that the tumor-suppressive effects of miR-128 are linked to its ability to inhibit CSCs by targeting key molecules, such as BMI-1, and Lo et al. mentioned that STAT1-IFIT5 plays a crucial role in PCSC acquisition by accelerating the turnover of specific microRNAs, such as miR-128 [118,141]. This evidence concerns the gene BMI1 and neoplasm.