As a receptor kinase, AXL was initially identified as a transforming gene in chronic granulocytic leukemia, but with deeper research, it has been gradually discovered that AXL is also able to participate in the regulation of a variety of responses in tumor cells, including cell proliferation, survival, and migration [73]. The gene discussed is AXL; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.