EGFR and disease arising from reactivation of latent virus: Modern understanding of different human oncogenic DNA viruses highlights several common biological features: establishing latent infection, dependency on the infected cell replication capacities, capability to disrupt the cell cycle, interfering with epithelial-mesenchymal transition (EMT)-/epidermal growth factor receptor (EGFR)-associated pathways, and influencing the Wnt/β-catenin pathway [1].