Further, a study comparing other CPT isoforms (CPT1A, CPT1B, and CPT2) concluded that except for CPT1C, other members of the CPT family have not been found to be associated with cancer cell senescence for the time being, and suggested a potential molecular mechanism by which CPT1C/c-Myc/p27 contributes to the induction of cellular senescence by CPT1C knockdown [115]. The gene discussed is CPT1A; the disease is cancer.