The progression of endometriosis is impacted by specific anti-inflammatory cytokines, including interleukins (IL) IL-1 beta, IL-5, IL-6, IL-7, IL-12, IL-4, and IL-10, which contribute to the persistence, expansion, infiltration, differentiation, angiogenesis, and immune evasion of endometriotic lesions [36]. This evidence concerns the gene IL1B and endometriosis.