ERBB2 and neoplasm: The increased presence of interactions in cytobands 17q11.2 and 8q24.3 in Her2 and Basal subtypes underscores the shared genomic vulnerabilities of these subtypes, as both regions are linked to critical oncogenes like ERBB2 and MYC, whose amplification exacerbates genomic instability, promoting resistance to therapy and aggressive tumor progression [44,45].