In rat airway SMCs, the upregulated lncRNA TUG1 and GAS5 act as sponges of miR138-5p and miR10a, respectively, and the lncRNA BCYRN1 increases the stability of the transient receptor potential 1 (TRPC1) protein, thereby promoting the proliferation and migration of airway SMCs in asthma [157,158,159]. This evidence concerns the gene TUG1 and asthma.