CSF1R and persistent truncus arteriosus: Another study using Cite-Seq, scRNASeq, and CytOF technologies to discriminate resident from infiltrating macrophages found that it is the resident macrophage population that readily expands one week following TAC injury and that timely regulated blockade by colony stimulating factor 1 receptor 1 (CSFR1), which preferentially abolishes resident macrophages early after TAC, leads to extended fibrosis 6 weeks later [88].