LGALS3 and cancer: Often overexpressed by cancer cells is N-acetyllactosamine (LacNAc), interacting with galectins (e.g., galectin-3 binding to Lac, LacNAc or poly-N-acetyllactosamino glycan) and contributing to carcinogenetic events, such as metastases’ formation and cancer-related adhesion or immune escape [147,148].