Table 8 shows that the biological processes enriched with the downregulated DEGs were DNA replication (p = 0.003), tyrosine metabolism (p = 0.006), metabolism of drugs (p = 0.003), the intestinal immune network for immunoglobulin A production (p = 0.009), TGF-β signaling pathway (p = 0.007), and amoebiasis (p = 0.0056). On the other hand, Table 9 shows that DEGs upregulated the biological processes of non-alcoholic fatty liver disease (p = 0.00003), oxidative phosphorylation (p = 0.0033), and chemical carcinogenesis–ROS (p = 0.0016). This evidence concerns the gene TGFB1 and metabolic dysfunction-associated steatotic liver disease.