Mir-3130 has been found to inhibit the expression of NDUFS1 to promote the invasiveness of lung adenocarcinoma in vivo and in vitro [33] and could also regulate the miR-3130-3p/NFYA/SATB1 axis to promote the occurrence and development of endometrial cancer cells [34]. This evidence concerns the gene NFYA and lung adenocarcinoma.