Additionally, other research has shown that FKBP51 can activate mitophagy by suppressing PPAR‐γ after multiple sclerosis.[60] Furthermore, FKBP38 recruits LC3A to mediate Parkin‐independent mitophagy[61] and prevents apoptosis during the mitophagy process.[62, 63] Hence, the regulatory effects of tacrolimus on other FKBPs warrant further investigation. The gene discussed is MAP1LC3A; the disease is multiple sclerosis.