NFKB1 and Sepsis: Animal experiments have shown that beta‐sitosterol can significantly suppress the inflammatory response by inhibiting the expression levels of tumour necrosis factor‐α(TNF‐α) and NF‐κBi through the inhibition of the NF‐κB pathway, as well as reduce the levels of AST and ALT in the serum while increasing the GSH content in the liver, thereby alleviating the liver oxidative stress response and preventing organ dysfunction associated with sepsis.36