CSNK1E and colorectal carcinoma: Here we observed that PRRC2A expression was upregulated in CRC by transcription factor ATF1, and in turn, elevated PRRC2A directly increases the stability of CSNK1E mRNA (encoding CK1ε) in an m6A‐dependent manner, whereas CK1ε concomitantly activates both WNT and YAP signaling pathways, thus promoting CRC progression.