N6‐methyladenosine (m6A) modification is the most prevalent internal modification in eukaryotic mRNAs, and its dysregulation plays a critical role in promoting cancer progression.[3, 4, 5] m6A modifications of target RNAs need to be recognized by m6A reader proteins to regulate RNA export, splicing, translation, and degradation.[3, 6] Several m6A reader proteins have been identified,[3] including YTHDF1‐3, IGF2BP1‐3, and YTHDC1‐2. The gene discussed is YTHDF1; the disease is cancer.