Our findings revealed an augmentation in the myeloid CD11c+ populations, including MΦ (F4/80 + MHCII+ CD11c+) and DCs (F4/80− MHCII+ CD11c+), within the fibrotic lungs of bleomycin‐treated mice, exhibiting a correlation with the progression of pulmonary fibrosis. The gene discussed is ITGAX; the disease is pulmonary fibrosis.