These observations contradictprevious MD studies, suggesting that SK9 could stabilize amylin inits native conformation and prevent its misfolding and self-assembly.By dispelling the possibility of direct binding of the viral envelopeprotein or its fragments to full-length amylin, the experimental datapresented in this study may help narrow the spectrum of potentialmechanisms by which SARS-CoV-2 infection can induce type 2 diabetes. The gene discussed is IAPP; the disease is type 2 diabetes mellitus.