Recent studies have suggested that the combination of tumor morphology and a unique functional chimeric protein, formed by the in-frame fusion of DNAJ homolog, subfamily B, member 1 (DNAJB1), and the catalytic subunit of protein kinase A (PRKACA) genes on chromosome 19, is thought to be specific to FLC and may play a role in its pathogenesis [6,8]. Here, DNAJB1 is linked to neoplasm.