Alcohol consumption impairs gut barrier functions, which consequently increases circulating endotoxemia and aggravates alcoholic steatohepatitis.[25] In the current work, the LJ or HKLJ treatments significantly reversed the negative effects of ethanol feeding, as indicated by a notable increase in the levels of lipopolysaccharide (LPS) and pro‐inflammatory cytokines (TNFα, IL‐6, and IL‐1β) in the circulation of mice (Figure 2A–D). Here, IL6 is linked to alcoholic fatty liver disease.