TGF‐β reportedly downregulates STING expression, and STING supports IFN‐β and CCL5 levels.[25] TGF‐β exhibited limited effects on cGAS or cGAMP levels,[25] suggesting that increased levels of TGF‐β in the late tumor stage might induce STING downregulation. The gene discussed is IFNB1; the disease is neoplasm.