ANXA2 and acute lymphoblastic leukemia: Survival extension in BCR-ABL1+ B-ALL was partially due to reduced LIC homing to an ANXA2 KO BMM, but incomplete rescue of B-ALL in intrafemorally transplanted ANXA2 KO mice and more pronounced prolongation of B-ALL survival in non-irradiated recipients suggest major contributory roles of an ANXA2-deficient BMM for B-ALL progression.