Though the MD value was determined by the meta-analysis and construction of a forest plot in a random effects model, it was found that CRP was significantly higher among the myocarditis patients (6.03; 95% CI: 2.41-9.64) compared to the control participants indicating increased production and persistence of CRP in response to the immune activation due to cardiac cellular damage and inflammation (Figure 3). Here, CRP is linked to myocarditis.