In support of enhanced SCD1 activity, post-mortem AD brains and triple-transgenic AD (3xTg-AD) mice demonstrate an accumulation of MUFA-enriched TGs within ependymal cells in the subventricular zone (SVZ) lining the ventricles, with the majority of TGs being enriched in SCD1-derived MUFAs such as OA [78]. This evidence concerns the gene SCD and Alzheimer disease.