Although these findings align with previous observations showing that (1) absence of SCD1 heightens splenic follicular Tregs after influenza immunization [49], (2) ATGL favors the hydrolysis of TGs and phosphatidylcholine rich in PUFAs [50], (3) DHA is a potent endogenous ligand for PPARγ [51], and (4) both DHA and PPARγ promote Treg differentiation and suppress neuroinflammation [51–58], several unanswered questions persist. This evidence concerns the gene PPARG and influenza.