ZFP36 and posterior cortical atrophy: Wound closure of Ptenf/fZfp36f/f cells occurred significantly faster than that of Ptenf/f cells, but was slower in comparison with a previously described metastatic, neuroendocrine PCa model, Ptenf/fRb1f/f (32), which hints that loss of Zfp36 may result in an intermediate phenotype, poised to become aggressively metastatic.