Specifically, we report here that CLP-sepsis caused a significant increase in IL-1β, IL-6, CCL3, CLL4, CXCL1, CXCL10 and GM-CSF, all of which contribute to sepsis-induced cytokine storm and, hence, to cardiac and organ dysfunction (17, 90–95). Here, CXCL1 is linked to Sepsis.