manages NAFLD involving the regulation of several key factors, including c‐Jun N‐terminal kinases, sterol regulatory element‐binding protein‐1c, carbohydrate response element binding proteins, peroxisome proliferator‐activated receptors (PPARs), and cytochrome P450.[20] One of the major tanshinones isolated from Salvia miltiorrhiza Bge, dihydrotanshinoneI(DhT), has been shown to have cardiovascular protective, anti‐inflammatory, anti‐oxidant, and hepaprotective actions.[21] However, the therapeutic effect and mechanism of DhT in treating NAFLD are still unknown. The gene discussed is MLXIPL; the disease is metabolic dysfunction-associated steatotic liver disease.