We further predicted cellular interactions between DC2, moDC_3 and malignant T cells via the ligand–receptor pairs CD58–CD2, which have previously been reported to mediate cancer immune evasion49, and putative interactions via LGALS9–HAVCR2 (also known as TIM3), LGALS9–P4HB and TNFSF12–TNFRSF25 (Fig. 6f). The gene discussed is LGALS9; the disease is cancer.