CD58 and cancer: We further predicted cellular interactions between DC2, moDC_3 and malignant T cells via the ligand–receptor pairs CD58–CD2, which have previously been reported to mediate cancer immune evasion49, and putative interactions via LGALS9–HAVCR2 (also known as TIM3), LGALS9–P4HB and TNFSF12–TNFRSF25 (Fig. 6f).