Increased cTnT may be related to microvascular coronary disease or unnoticed cardiomyocyte injury observed in conditions such as coronary artery disease, diabetes mellitus, chronic kidney disease, as well as to left-ventricular (LV) strain, decreased subendocardial perfusion, endothelial dysfunction and apoptosis in the setting of heart failure (HF)6. Here, TNNT2 is linked to hydrops fetalis.