Similar tumor-suppressive activities of S. aureus have also been observed in glioblastoma, where the presence of intracranial S. aureus in several patients were associated with longer survival.(37) Future research is required to investigate whether the presence of Staphylococcus or S. aureus in TNBC influences responses to immunotherapy, as well as to characterize the functions of Staphylococcus in ER+/PR+ and other BC subtypes. The gene discussed is ESR1; the disease is neoplasm.