For GFAP, a significant positive contribution to the interaction was observed in the hippocampus of both the dementia and no-dementia groups (HIP, β = 43.59, p = 0.022 and β = 24.852 and p = 0.001, respectively) and in the temporal neocortex of the no-dementia group (TCx, β = 13.873 and p < 0.001). The gene discussed is GFAP; the disease is dementia.