Overexpression of a RAD51-interacting domain of BRCA2 has been shown to disrupt DNA repair by homologous recombination in human cultured cells.31,32 Successful meiosis requires suppression of S phase during the second round of cell division.33 Notably, overexpression of GZF134, YWHAZ35, ACSL336, ZDHHC1637, or COX7A2L38 stimulates mitosis in human immortalized cells and mouse cancer models. Here, RAD51 is linked to cancer.