reported that in an experimental autoimmune encephalomyelitis (EAE) model of MS, TNF receptor 2 (TNFR2) ablation in microglia caused early onset of EAE with increased immune cell infiltration and activation and demyelination, whereas TNFR2 ablation in monocytes/macrophages had opposite effects [38]. Here, TNFRSF1B is linked to experimental autoimmune encephalomyelitis.