The results suggest that H19 acts as a ceRNA for AQP1 by sequestering miR-874, highlighting its potential as a therapeutic target for mitigating sepsis-induced myocardial dysfunction (54).In a bacterial endotoxin-induced mouse model, deletion of aquaporin 9 (AQP9) improved survival and reduced oxidative stress. This evidence concerns the gene AQP1 and Sepsis.