While treatment options for the two subtypes of NSCLC, namely LSCC and LUAD, were previously similar (14), LSCC exhibits a higher prevalence of certain mutated genes, including tumor protein p53, glutamate receptor, metabotropic 8, Erb-B2 receptor tyrosine kinase 4, Kelch-like ECH-associated protein 1 (KEAP1), and mucin 16 (15). Here, KEAP1 is linked to non-small cell lung carcinoma.