To demonstrate that the observed IL22-dependent phenotype was due to CD4+ TH17 cells rather than ILC3s, the authors analyzed caecal LP CD4+ LTi cells from GclcfloxCD4Cre and Gclcflox mice at days 4 and 7 post infection revealing no defect in LTi cell frequency and cytokine production in contrast to IL17+IL22+ TH17 cells. Here, IL22 is linked to infection.