CPZ provokes the mitochondrial dysfunction of myelin-forming cells, impairs oligodendrocyte function, induces demyelination in several brain regions, and has been found to nearly resemble “pattern-III lesions” in MS-patients, from the histopathological aspect (Clarner et al. 2012; Nicola et al. 2024). The gene discussed is CPZ; the disease is myeloid sarcoma.