Further analysis of gene module scores for cytotoxicity, divided between cells from controls and children with CHD, suggests an increased expression for genes encoding cytotoxic effector molecules (e.g., GZMA, GZMB, TNFα or IFNγ) of Vδ1 T cells (c1) in children with CHD (Fig. 4g). The gene discussed is IFNG; the disease is coronary artery disorder.