TGFB1 and Duchenne muscular dystrophy: This model appears to more closely mimic human pathology, exhibiting a more severe pro-fibrotic and pro-inflammatory phenotype due to polymorphisms in the Latent Transforming Growth Factor Beta Binding Protein 4—LTBP4 gene [17–19], a Transforming Growth Factor Beta 1—TGF-β key regulator, also detected in DMD patients [7, 8].