METTL3 and cancer: It has been confirmed by research that inhibiting HDAC6 can partially restore the cell phenotype with decreased cilia length.[36] Moreover, HDAC6 has been extensively reported to induce ciliary resorption in cancer cells.[35, 37] In our data, we found that overexpression of HDAC6 predominantly rescued stimulation implication of METTL3 depletion on the cilia assembly and elongation in HeLa and SiHa.