It has been confirmed by research that inhibiting HDAC6 can partially restore the cell phenotype with decreased cilia length.[36] Moreover, HDAC6 has been extensively reported to induce ciliary resorption in cancer cells.[35, 37] In our data, we found that overexpression of HDAC6 predominantly rescued stimulation implication of METTL3 depletion on the cilia assembly and elongation in HeLa and SiHa. Here, HDAC6 is linked to cancer.