Additionally, our analysis of patient samples revealed that the RNA level of MALAT1 was significantly decreased in the HBV-infected GCB-type DLBCL patients (Fig. 6E), while the SLC7A11 expression was increased (Fig. 6F), with a negative correlation observed between MALAT1 and SLC7A11 expression (Fig. 6G), further supporting the pivotal role of MALAT1 in modulating ferroptosis in the context of HBV-infected DLBCLs. This evidence concerns the gene SLC7A11 and diffuse large B-cell lymphoma.