To clarify that IL‐1β inhibition is similar to TNF‐α antagonism for suppression, they were surprised to find that IL‐1β deficiency in the CaCl2 model does not block M1 macrophage polarization and does not inhibit AAA formation, whereas TNF‐α deficiency tends to polarize them toward M2, reducing the destructive effects of M1 macrophages. Here, TNF is linked to triple-A syndrome.