PRMT1 and hepatocellular carcinoma: Plate cloning assays, CCK-8 assays and cell suspension assays showed that overexpression of PRMT1 promoted the colony-forming ability (Fig. 3A), cell viability (Fig. 3B) and stemness (Fig. 3C) of HCC cells, whereas overexpression of PRMT1 with concomitant interference of ME2 expression reversed this effect (Fig. 3A–C).