The pathological stages and patterns of tau accumulation following repetitive brain injury, mainly from sports injuries, have since been extensively characterised as ‘chronic traumatic encephalopathy’ (CTE) [10, 11, 12], and in epilepsy, CTE has been reported at post‐mortem in isolated poorly controlled cases [13, 14] and CTE neuropathological change (CTE‐NC) was also noted in samples taken during epilepsy surgery [15, 16]. The gene discussed is MAPT; the disease is epilepsy.