TGFB1 and acute lymphoblastic leukemia: In a different biological setting, BBR was shown to stimulate miR-24-3p expression in lymphoblastic leukaemia cells.33 Here, we show that BBR likewise increases miR-24-3p expression in TGFβ1-treated hMT and their secreted EVs while decreasing ECM protein secretion and interstitial fibrosis in this model.